Boronia

This weekend. $10 microchipping. https://petstockblitz.simplybook.me/v2/?fbclid=IwAR0VoyaS_z6Fc-G9GPFOD-p-kezt9uv0ukiEg5pCXc6pRcMcov9xsUjYBUI#book/category/12/

https://www.brisbanetimes.com.au/world/north-america/woman-reunited-with-missing-dog-12-years-later-and-1600km-from-home-20191015-p530o5.html Woman reunited with missing dog 12 years later and 1600km from home By Brittany Shammas October 15, 2019 — 1.25am Dutchess the fox terrier squeezed out the door at her owner's house in Orlando, Florida, one day in February 2007 and didn't return. A devastated Katheryn Strang made "lost dog" signs and took daily trips to the local shelter for months, desperate to find her. On Friday, Strang finally got the reunion she had been hoping for - 12 years after Dutchess disappeared. A man found the dog under his shed in the Pittsburgh area, almost 1600 kilometres from Orlando. He took the shivering animal to a shelter, Humane Animal Rescue. There, a scan of Dutchess's microchip linked her to Strang, who had since moved to Boca Raton, Florida. Katheryn Strang is reunited with her fox terrier, Dutchess.Credit:AP At the shelter on Friday, Strang cried as an employee handed her the dog she had always hoped would one day return to her. Staff members posted footage of the emotional reunion on Facebook, writing, "This is why we do what we do." "Dutchess. Hi, baby. I missed you," Strang says in the video as she strokes the dog's face. "Your face is all white." No one knows how Dutchess made the trek to Pittsburgh - or what she has been doing for the past 12 years. Strang joked that the dog could "tell me some stories." She said she couldn't imagine that Dutchess had walked the whole way. The dog, who is now 14, was hungry, shaking and "in serious need of a nail trim" when she arrived at the shelter, said Torin Fisher, an admissions counsellor with Humane Animal Rescue. She was also "a little nervous about the situation, which, who could blame her?" Fisher said. But otherwise, Dutchess was in pretty good shape. Shelter employees soon found the microchip - an implanted device that contains a unique number that can be traced back to an animal's owner. Seeing that Dutchess's owner lived in Florida, Fisher figured the family must have recently moved to Pittsburgh. Only when she called Strang did she learn "how incredible of a situation it actually was." Dutchess had been missing for 12 years.Credit:AP "The whole story kind of unfolded while we were on the phone," Fisher said. "And we were equally excited and shocked and surprised." Strang had always held out hope that Dutchess might be found, even after so many years. She paid a $US15 ($22) annual fee to keep the microchip active, saying on Friday that she would have continued that for years. Still, she was in disbelief as she drove to Pittsburgh. "Until she's in your arms, it's just gut-wrenching," she said. The Washington Post

Give her some cranberry powder as well rascal, I put a little powder in Zara's food each time (she is prone to struvite crystals) I buy the capsules and each capsule lasts around 5 days so that's 10 doses per capsule. I just cut the capsule end off and sprinkle. It may help and will not do any harm anyway. Also give her segments of oranges if she'll eat them, otherwise some vit C powder

and dad supervising dinnertime

it's lunch-time and i wonder what sort of bird this is, it looks like a juvenile something

Yes! Three chickies

She sure is Rozzzie. I checked this morning while mumma is still sleeping, there seems to be a un-hatched egg. And a little later when she has left to find breakfast there seems to be three little chicks...two heads and one hatched later and you can only see its bum-hole right at the front of the 3rd screen-shot

I had a look now and think the other eggs have hatched, maybe four chicks. 5.15 and it's dinner-time...

Hooo... breakfast

6.24

For those DOLers who saw the news two week's ago about that poor man, Simon, who was killed near Monto. His dog, Merlin, ran away after the crash and locals have been searching for her. Well she's been found: https://www.abc.net.au/news/2019-10-01/merlin-the-missing-dog-turns-up-after-a-fortnight-in-the-bush/11564308 Merlin the missing dog emerges from the bush a fortnight after owner's death By Jemima Burt and Rachel McGhee Updated about 10 hours ago Photo: Merlin appeared as if by magic after a fortnight in the bush. (ABC News: Rachel McGhee) Related Story: Search intensifies for Merlin the missing dog after owner killed in car crash A dog that bolted into southern Queensland bushland after surviving a car crash that killed its owner has been found alive, more than two weeks later. Key points: A local farmer had spotted Merlin and left out the dog's bedding with food Merlin was found lying on the bedding on Tuesday morning Simon Hannan's family say they are thrilled Merlin is safe and well Merlin the kelpie-cross had been travelling around Australia with its Victorian owner Simon Hannan, but he was killed by a car between Monto and Eidsvold, west of Bundaberg, last month. The distressed dog ran into the bush from the crash scene. After Mr Hannan died in hospital, the North Burnett community banded together in a bid to help his family track down the dog, the final link to their loved one. Hopes for Merlin's survival faded quickly last week, with the drought-affected area offering up challenging conditions for the domestic dog's survival. But for the past five days, Eidsvold farmer Bill Kerle has been tracking Merlin's paw prints on his property, which is only a couple of hundred metres from the crash site. Mr Kerle said he spotted Merlin on Monday morning, and decided to set up a trap for the dog near his dam. He placed the dog's bedding inside the trap, with some pork steaks, and found Merlin asleep inside the trap on Tuesday morning. "I nearly fell off my quad bike, I couldn't believe it," he said. Photo: Merlin with Simon, who was killed in a car crash near Monto. (Facebook: David McDougall) Shortly after Mr Hannan's death, his mother Jan Hannan and stepfather David McDougall travelled to the area from their home in Western Australia, but were back at home when they received a call telling them Merlin had turned up. "We believe he had put Merlin's bed out overnight and some food and she was sitting in it this morning when they found her," Ms Hannan said. "We're so thrilled to have his dog and know that she's safe and well." Ms Hannan said there are no words to describe losing a son, but that finding Merlin offered some comfort. "We can't bring my son back but we can find his dog and we can make sure that she now has a good life." Photo: Eidsvold farmer Bill Kerle managed to capture Merlin in a humane dog trap. (ABC News: Rachel McGhee) She said she was very grateful to the farmer who recovered Merlin. "He has our undivided gratitude. We will personally be talking to him, he's a very humble gentleman," Ms Hannan said. "He will not want public recognition but we will be doing something privately with him." The search for Merlin gained national attention after Mr McDougall posted about it on Facebook. Photo: Merlin was lured back to civilisation by bedding and food. (Supplied) It has since been shared more than 17,000 times, with people coming from all over to help. Mr McDougall said it had been a difficult fortnight. "We weren't expecting a good outcome," he said. "It may have taken 15 days, but Merlin has returned so there is always positives. "To have her recovered now is … all that stress is gone. We've had to deal with the tragedy of Simon and to be able to get total closure. Photo: Merlin ran from the scene of the crash that killed the dog's owner, Simon Hannan. (Supplied) "For all Simon's family and friends and everyone that was out there looking for Merlin, it's just a huge relief," Mr McDougall said. He said Merlin seems to be in good health. "She's not distressed, not in a bad condition — there's going to be a lot of pats and cuddles and dog treats in her foreseeable future." Mr McDougall thanked the people of Monto and Eidsvold for their help and hospitality. "The amount of support and encouragement, and advice has been truly outstanding," he said. "We stayed in the local pubs in Monto, they wouldn't let us pay for accommodation. "I just can't thank the community, Monto and Eidsvold [enough], they are fantastic people and we know they're having a raw deal at the moment with rainfall and that sort of stuff with the drought."

Thank you for the link Rebanne ...Zara's son, she is so proud of him https://my.rasv.com.au/Results/Competitions/2019-rms-dog/Classes/DADT11 https://my.rasv.com.au/Results/Competitions/2019-rms-dog/Classes/DADT2S https://my.rasv.com.au/Results/Competitions/2019-rms-dog/Classes/DADT6S

That's good news that she is fine and dandie Kazm, it's a pity there are no pups but maybe for the best and there is always a 'next time' then her owner will be more aware of what's needed and keep on top of things. I think some of the comments were, perhaps, harsh, but mostly that comes with concern for the dog so it's usually 'wade in, boots and all' with the hope that the OP will do something NOW. Maybe some of the posters need to tone it down a tad and try a gentler approach, we could chase members away with bossy attitudes.

it may be a plan for them to whizz her to the vets and check anyway Kazm then they, and you (and us) will find out for sure

I am so sorry you have had to say goodbye to your beautiful girl Sheena

link to Phys.org article here Team develops treatment for canine megaesophagus by University of Missouri-Columbia This image shows a canine patient during a video fluoroscopic swallow study. These studies, performed by members of a collaborative research project at the University of Missouri, were a major component of revolutionary techniques developed for the diagnosis and treatment of megaesophagus, a devastating disease of dogs. Credit: MU College of Veterinary Medicine The Veterinary Health Center (VHC) at the University of Missouri's College of Veterinary Medicine is pioneering a new approach to treat one type of canine megaesophagus, a devastating disease of dogs. A partnership between the VHC's Small Animal Internal Medicine, Radiology, Surgery, and Nutrition services and an investigator in the Otolaryngology-Head and Neck Surgery department at the university's School of Medicine has resulted in the discovery of a breakthrough treatment for a subpopulation of dogs with megaesophagus. The Mizzou team has identified a defect of the lower esophageal sphincter (LES) as a potential treatable cause of megaesophagus. Megaesophagus (ME) refers to a large, dilated esophagus with poor or no motility preventing normal passage of food and liquid into the stomach. With ingesta not reaching the stomach to produce the sensation of being full, the dog will continue to eat. As a result, the esophagus enlarges greatly. Dogs end up not getting enough calories so they waste away. Dogs with ME also regurgitate large amounts of undigested food and some of that material can be inhaled into the lungs. This inhalation can result in aspiration pneumonia, a dangerous additional symptom that kills many affected animals. "In general, dogs with megaesophagus typically die of malnutrition, aspiration pneumonia, or are euthanized because the owners are told they have a terrible prognosis," according to Associate Professor Carol Reinero, DVM, PhD, an internal medicine specialist helping coordinate the efforts of the multidisciplinary team. "We're taking a closer look at a subpopulation of dogs with idiopathic megaesophagus, which means we don't know what causes it. We run all of our traditional tests, but we've never been able to find an underlying cause. Now with our video fluoroscopic swallow studies, we have identified an abnormality that we believe is driving the problem: an LES-achalasia-like syndrome." The LES acts as a valve between the esophagus and the stomach, opening when food and water are swallowed, then clamping tight so food doesn't come back from the stomach into the esophagus. In dogs afflicted with megaesophagus caused by an achalasia-like syndrome, the LES remains closed. "Video fluoroscopic swallow studies have been around a long time," Reinero said. "The problem with prior protocols is they did not represent normal eating and drinking behaviors. Those tests had very little to do with reality. A dog had to be restrained, lying on its side, and syringe-fed barium, which was not palatable even when mixed with food. If dogs don't eat during the swallow study you're not going to get a diagnostic study." In order to receive a better diagnostic result, the Mizzou collaborative research team blended innovation with adaptation, developing new techniques and tools while borrowing procedures from human medical practices to diagnose and treat this type of canine ME. Assistant Professor Teresa Lever, MS, PhD, from the Otolaryngology-Head and Neck Surgery department, spearheaded the development of trapezoidal holding chambers or kennels—which are now patented—where dogs walk in and are funneled to the narrow end of the enclosure. The animals are more naturally restrained and are given food that they consume while standing upright. Lever's lab, in cooperation with an independent company that conducts feeding trials in dogs, also developed a number of different recipes and consistencies of food and liquid to optimize how well the dogs could take it down and discover which contrast agent was more palatable and effective. "So, now we have these chambers, we have optimized palatable recipes, and we have experience in normal, healthy dogs without swallowing disorders, as well as lots and lots of patients coming in with other swallowing disorders," Reinero said. "It was in the process of developing this protocol and having clinical patients come in when we recognized that LES-achalasia-like syndrome was the underlying cause of megaesophagus in some dogs. That's when we began to adapt what they do with humans, a therapy where we're opening or dilating the LES with Botox and/or balloon dilation. "We perform an endoscopy to first dilate the LES and then administer Botox, which paralyzes the sphincter muscles that formerly wanted to remain closed. While we are still evaluating this procedure, we've had dogs with remarkable clinical improvement. Additionally, when we repeat the fluoroscopic studies, we can document an open LES. The patients that show improvement can be candidates for surgery, and that surgery is potentially curative," Reinero said. During a patient's recovery, residents in the Nutrition Service monitor the dog's weight and food intake. The amount of food an owner may give a pet might need to be altered every couple of days. James Schachtel, Bvet Med, MRCVS, is a Radiology resident and key collaborator in the study. "This approach gives these dogs a chance, whereas a lot of them didn't have much of one," Schachtel said. "At this time, it is early in the evaluation process, but it's a novel approach that shows promise. "This subpopulation can receive a really significant benefit from our direct ability to detect their malady. It can give them a really good quality of life. This is a revolutionary diagnostic technique for a disorder identified with a pathological outcome. It offers us the opportunity to use therapies that have been successful in people, so we're optimistic we can experience similar success with canines," Schachtel said. Schachtel will present an oral abstract of the swallowing protocol and early identification of the LES achalasia on Oct. 20 at the American College of Veterinary Radiation Annual Scientific Conference in Orlando.

Bedlington Benches September 4, 2019 National Purebred Dog Day® In Northumberland, England in a county situated in North East England is a former mining town named Bedlington, and yes, it’s the town from which the terrier got its name. This is a very old town. Bedlington and the hamlets belonging to it were bought by Cutheard, Bishop of Durham between 900 and 915, and Bishop Walcher was granted royal rights by William the Conqueror. The breed didn’t get it’s current name until 1825, and it’s said that some Bedlington Terriers have pedigrees that can be traced back as far as 1782. In fact, the first dog show that offered a class for Bedlington Terriers was held in 1870 in the town of (wait for it) Bedlington! Bedlington is mighty proud of its native son, so to speak. It has a North League Soccer team that is called The Terriers, and the town has even installed park benches in the form of Bedlington Terriers: A photo of one of these benches also graces the West Bedlington Town Council’s Facebook page. On Front Street West in Bedlington is the current Red Lion Inn that was (re)built in 1902 as a replacement for the inn of the same name which stood on the same site. Outside the pub is another bench inspired by the Bedlington Terrier: Inside the pub is a wire sculpture by Gary Tiplady, an artist and local celebrity who, at 7’3” tall, made use of his large hands to bend and shape sheets of wire into life-like animals like the Bedlington piece named, “Give a Dog a Bone.” Not every owner can manage to have their own Bedlington Terrier park bench, but Spoonflower, the fabulous textile marketplace, makes it easy for you to have anything fabric related in the breed: Now there’s no excuse not to have your curtains, party frock, bow tie, or dog cushions reflect your affection for the breed! This entry was posted in Bedlington Terrier and tagged Bedlington Terrier, fabric, park bench.

this is the dry food she is eating in the morning: https://www.petstock.com.au/product/dog/royal-canin-veterinary-urinary-s-o-dry-dog-food/58497 and a list of the ingredients, as you see they are pretty crappy, no wonder her skin is going black...an awful amount of corn, fillers and sodium to make her wee more often therefore drinking more to flush the kidneys Rice, maize flour, animal fats, dehydrated poultry meat, maize gluten, minerals, hydrolysed animal proteins, vegetable fibres, soya oil, fish oil, fructo-oligo-saccharides, marigold extract (source of lutein). ADDITIVES (per kg): Nutritional additives: Vitamin A: 16200 IU, Vitamin D3: 1000 IU, E1 (I ron): 52 mg, E2 (Iodine): 5.2 mg, E4 (Copper): 10 mg, E5 (Manganese): 67 mg, E6 (Zinc): 202 mg, E8 ( Selenium): 0.08 mg - Preservatives - Antioxidants. ANALYTICAL CONSTITUENTS: Protein: 18% - Fat conte nt: 17% - Crude ash: 6.6% - Crude fibres: 2.3% - Calcium: 0.5% - Phosphorus: 0.5% - Sodium: 1.2% - Chloride: 2.21% - Potassium: 0.8% - Magnesium: 0.05% - Sulphur: 0.3% - Urine acidifying substances: calcium sulphate (0.55%), DL-methionine. If I can't find a low magnesium kibble I will just feed her chicken and VAN Complete mix, I am feeding kibble in the morning so she can have some vitamins and minerals that she may miss out on if fed just chicken and VAN and it's more of a convenience as mornings are busy.

I am going to take her off the Royal Canin and see if the blackness on her skin clears up, she can have chicken and VAN, golden paste, cranberry powder and vitamins instead.

I checked and they appear to have it just for cats, perhaps I'll try the Hills though I'm not too sure if it's all it's cracked up to be, it appears, from some comments I read a while back by Dolers, to make the dog drink more which flushes out the kidneys, seems a odd thing to do. I'll have a look at the sodium content later on today

My dog is fed Royal Canin Veterinary Urinary S/O Dry Dog Food at breakfast time, for tea-time she gets raw chicken (chopped up drumstick or wings and 1/4 frame) with Vets All Natural Complete Mix Adult Senior mixed in. She did have bladder stones/struvite crystals (that's why the Urinary S/O) and suffers from some itchiness on her haunches; she licks them and her bum as well, her anal glands were expressed but she still licks. After her struvite crystals diagnosis and treatment there have been none detected in her urine. She was on Niralone to see if it helped her licking/itchy problem which it did, but her vet took her off those tablets as the skin on her back is becoming dark and there was hair loss, she stopped her licking for around one week but then started again after discontinuing them, we have been trying 1/4 Apoquel morning and night for two weeks then gradually cutting the dosage back, not so much licking now but her skin is still blackish, though only on her back...her tummy, chest, head and legs are nice and pink. I thought she may be extra-sensitive to the Canin Veterinary Urinary and wondered if there is an alternative. She must be kept on a low low low magnesium diet as we want to prevent struvite crystals forming again. Any suggestions will be very helpful

Happy Birthday for next week Miss Bolt. As Perse says...I hope you get cake and perhaps find a birthday rat somewhere Maybe mummy will post all your birthday piccies though I'm sure you won't look as lovely as you are in today's pic